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Dr.
D. Karunagaran is heading the Department of Cancer Biology at the
Rajiv Gandhi Centre for Biotechnology, Trivandrum. He has more than 35
international publications and written 3 book chapters and has guided 4
PhD students and 2 research associates. He is a member of several
international and national professional bodies and has received the CR
Krishnamurti Lecture award for the year 2003 from the Scoety of Biological
Chemists (India). Dr. D. Karunagaran did his MSc Bichemistry in JIPMER,
Pondicherry in 1979 and was appointed the same year as a Lecturer in
Biochemistry at the PSG College of Arts and Science, Coimbatore where he
continued to teach students till 1992. After his PhD in Biochemistry from
Sri Krishnadevaraya University, Anantapur, he went to Israel to do his
postdoctoral research at the Weizmann Institute of Science and then to MD
Anderson Cancer Center, Houston, USA. He has made significant
contributions in the field of signal transduction, mediated by
epidermal growth factor receptors (EGFRs) and their ligands implicated in
the development of human cancer. Dr. Karunagaran joined the Rajiv Gandhi
Centre for Biotechnology, Thiruvananthapuram in December 1997. He has
shown that the transcription factor, nuclear factor-kappa B (NF-kB)
is constitutively activated in high-grade squamous intraepithelial lesions
and squamous cell carcinomas during human cervical cancer progression. Dr.
Karunagaran has proposed that differential over expression of certain
antiapoptotic proteins may account for the failure of curcumin to induce
apoptosis in some cancer cells. Results from his laboratory suggest
antiapoptotic roles for NF-kB,
Bcl-XL and heat shock proteins such as hsp70 in curcumin-induced
apoptosis. The laboratory has identified that NF-kB
can also regulate epidermal growth factor-induced apoptosis by a similar
mechanism. His laboratory has also reported a novel Smad 2 mutation
in a human cervical tumor sample and loss of expression of Smad 2
and Smad 4 in different stages of cervical cancer progression and
also the mutations of Smad 2 and Smad 4 in two of the six
human cervical cancer cell lines analyzed revealing an important role for
Smads in human cervical cancer for the first time.
List of the most important publications of Dr.
DEVARAJAN KARUNAGARAN
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Ramachandran Rashmi, Santhosh Kumar and Devarajan Karunagaran (2005)
Human colon cancer cells lacking Bax resist curcumin-induced apoptosis
and Bax requirement is dispensable with ectopic expression of Smac or
downregulation of Bcl-XL Carcinogenesis 26, 713-723
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Smitha, V. B., Vinesh Kumar, T. P., Deepti, A., Nair, A., Karunagaran,
D., and Anto, R. J. (2005). Sensitization of taxol-induced apoptosis by
curcumin involves down-regulation of nuclear factor-kappa B and the
serine/threonine kinase Akt and is independent of tubulin
polymerization. J. Biol. Chem., 280, 6301-6308
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Tessy T Maliekal, Ruby John Anto and Devarajan Karunagaran (2004)
Differential Activation of Smads in HeLa and SiHa Cells that Differ in
their Response to Transforming Growth Factor-β. J. Biol. Chem.,
279: 36287-36292
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Tessy T Maliekal, Marie-Lue Antony, Asha Nair, Ramasamy Paulmurugan and
Devarajan Karunagaran (2003) Loss of expression, and mutations of
Smad 2 and Smad 4 in human cervical cancer. Oncogene
22, 4889-4897
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Ruby John Anto, Manickam Venkatraman, and Devarajan Karunagaran (2003)
Inhibition of NF-κB sensitizes
A431 Cells to EGF-induced apoptosis whereas its activation by Ectopic
expression of RelA confers resistance J. Biol. Chem.
278, 25490-25498
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Asha Nair, Manickam Venkatraman, Tessy T. Maliekal, Balaraman Nair, and
Devarajan Karunagaran (2003) Nuclear Factor-κB
is Constitutively Activated in High grade Squamous Intraepithelial
Lesions and Squamous Cell Carcinomas of the Human Uterine Cervix.
Oncogene 22, 50-58.
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Anto, R. J., Maliekal, T. T., and Karunagaran D. (2000) L-929 cells
harboring ectopically expressed RelA resist curcumin-induced apoptosis
J. Biol.Chem. 275, 15601-15604
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Shao, R., Karunagaran, D., Zhou, B. P., Li, K., Lo, S-S., Deng, J.,
Chiao, P., and Hung, M. C. (1997) Inhibition of nuclear factor -
κB activity is
involved in E1A-mediated sensitization of radiation-induced apoptosis.
J. Biol. Chem., 272, 32739-32742.
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Karunagaran, D., Tzahar, E., Beerli, R. R., Chen, X., Graus-Porta, D.,
Ratzkin, B., Seger, R., Hynes, N.E. and Yarden, Y. (1996) ErbB-2 is a
common auxiliary subunit of NDF- and EGF-receptors: implications for
breast cancer. EMBO.J.15: 254-264.
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Karunagaran, D., Tzahar, E., Liu, N., Wen, D. and Yarden, Y. (1995) Neu
Differentiation Factor Inhibits EGF Binding: A Model for
Trans-Regulation Within the ErbB Family of Receptor Tyrosine Kinases.
J. Biol. Chem., 270, 9982-9990.
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